Spatial Vectorcardiography in the Wolff‐Parkinson‐White Syndrome: Correlation with Epicardial Mapping Findings
Identifieur interne : 000073 ( Main/Corpus ); précédent : 000072; suivant : 000074Spatial Vectorcardiography in the Wolff‐Parkinson‐White Syndrome: Correlation with Epicardial Mapping Findings
Auteurs : Kewal K. Talwar ; Per Blomström ; Nils Edvardsson ; Göran William-Olsson ; S. Bertil OlssonSource :
- Pacing and Clinical Electrophysiology [ 0147-8389 ] ; 1984-11.
English descriptors
Abstract
The spatial vectorcardiograms (VCG) of 13 patients with WPW syndrome due to single accessory pathways were analyzed and correlated with the excitation analysis obtained on epicardial mapping. The azimuth angle of the initial 10 ms cardiac vector was > + 90° (directed right and anteriorly) in patients with a left ventricular free wall; it ranged between 0° to 90° (left and anteriorly) in those with a left or right paraseptal free wall and was ‐ 30° (left and posteriorly) in one patient with a right ventricular free wall location. The elevation angle of the initial 10 and 20 ms cardiac vector was either zero or positive (inferiorly directed) in those with right and left ventricular free wall pathway. Among six patients with a paraseptal location, the elevation angle was negative (superiorly directed) in four and positive in two. Both the patients with a clockwise inscription of a QRS loop in the horizontal plane (HP) had pathways located to the left ventricle. Among the paraseptal group, at surgery, the accessory pathway could not be excised in two in spite of dissection very close to the IV (interventricular) septum. The elevation angle in both these patients was markedly negative (‐45° and ‐62°) in contrast to the other in whom surgical excision was successful. We thus conclude: 1) an azimuth angle of the initial 10 ms vector of more than 90° indicates early activation in the left ventricular free wall and less than 0° on the right ventricular free wall; 2) presence of a negative elevation angle [superiorly directed) of the initial 10 and 20 ms vector suggests a paraseptal location; 3) a markedly negative elevation angle may further indicate that the accessory connection is located very close to, or in, the IV septum.
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DOI: 10.1111/j.1540-8159.1984.tb05648.x
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I, S‐413 45 Göteborg, Sweden</mods:affiliation></affiliation></author><author><name sortKey="William Lsson, Goran" sort="William Lsson, Goran" uniqKey="William Lsson G" first="Göran" last="William-Olsson">Göran William-Olsson</name><affiliation><mods:affiliation>Division of Thoracic Surgery, Sahlgrenska Hospital, S‐413 45 Göteborg, Sweden</mods:affiliation></affiliation></author><author><name sortKey="Olsson, S Bertil" sort="Olsson, S Bertil" uniqKey="Olsson S" first="S. Bertil" last="Olsson">S. Bertil Olsson</name><affiliation><mods:affiliation>Division of Cardiology, Med. Clin. 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The azimuth angle of the initial 10 ms cardiac vector was > + 90° (directed right and anteriorly) in patients with a left ventricular free wall; it ranged between 0° to 90° (left and anteriorly) in those with a left or right paraseptal free wall and was ‐ 30° (left and posteriorly) in one patient with a right ventricular free wall location. The elevation angle of the initial 10 and 20 ms cardiac vector was either zero or positive (inferiorly directed) in those with right and left ventricular free wall pathway. Among six patients with a paraseptal location, the elevation angle was negative (superiorly directed) in four and positive in two. Both the patients with a clockwise inscription of a QRS loop in the horizontal plane (HP) had pathways located to the left ventricle. Among the paraseptal group, at surgery, the accessory pathway could not be excised in two in spite of dissection very close to the IV (interventricular) septum. The elevation angle in both these patients was markedly negative (‐45° and ‐62°) in contrast to the other in whom surgical excision was successful. We thus conclude: 1) an azimuth angle of the initial 10 ms vector of more than 90° indicates early activation in the left ventricular free wall and less than 0° on the right ventricular free wall; 2) presence of a negative elevation angle [superiorly directed) of the initial 10 and 20 ms vector suggests a paraseptal location; 3) a markedly negative elevation angle may further indicate that the accessory connection is located very close to, or in, the IV septum.</div></front></TEI><istex><corpusName>wiley</corpusName><author><json:item><name>KEWAL K. TALWAR</name><affiliations><json:string>Division of Cardiology, Med. Clin. I, S‐413 45 Göteborg, Sweden</json:string></affiliations></json:item><json:item><name>PER BLOMSTRÖM</name><affiliations><json:string>Division of Cardiology, Med. Clin. I, S‐413 45 Göteborg, Sweden</json:string></affiliations></json:item><json:item><name>NILS EDVARDSSON</name><affiliations><json:string>Division of Cardiology, Med. Clin. I, S‐413 45 Göteborg, Sweden</json:string></affiliations></json:item><json:item><name>GÖRAN WILLIAM‐OLSSON</name><affiliations><json:string>Division of Thoracic Surgery, Sahlgrenska Hospital, S‐413 45 Göteborg, Sweden</json:string></affiliations></json:item><json:item><name>S. BERTIL OLSSON</name><affiliations><json:string>Division of Cardiology, Med. Clin. 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The elevation angle in both these patients was markedly negative (‐45° and ‐62°) in contrast to the other in whom surgical excision was successful. 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Clin. I, S‐413 45 Göteborg, Sweden</affiliation></author><author><persName><forename type="first">PER</forename><surname>BLOMSTRÖM</surname></persName><affiliation>Division of Cardiology, Med. Clin. I, S‐413 45 Göteborg, Sweden</affiliation></author><author><persName><forename type="first">NILS</forename><surname>EDVARDSSON</surname></persName><affiliation>Division of Cardiology, Med. Clin. I, S‐413 45 Göteborg, Sweden</affiliation></author><author><persName><forename type="first">GÖRAN</forename><surname>WILLIAM‐OLSSON</surname></persName><affiliation>Division of Thoracic Surgery, Sahlgrenska Hospital, S‐413 45 Göteborg, Sweden</affiliation></author><author><persName><forename type="first">S. BERTIL</forename><surname>OLSSON</surname></persName><note type="correspondence"><p>Correspondence: Address for reprints: S. Bertil Olsson, M.D., Division of Cardiology, Dept. of Medicine I, Sahlgrenska Hospital, S‐413 45 Göteborg, Sweden</p></note><affiliation>Division of Cardiology, Med. Clin. I, S‐413 45 Göteborg, Sweden</affiliation></author></analytic><monogr><title level="j">Pacing and Clinical Electrophysiology</title><idno type="pISSN">0147-8389</idno><idno type="eISSN">1540-8159</idno><idno type="DOI">10.1111/(ISSN)1540-8159</idno><imprint><publisher>Blackwell Publishing Ltd</publisher><pubPlace>Oxford, UK</pubPlace><date type="published" when="1984-11"></date><biblScope unit="volume">7</biblScope><biblScope unit="issue">6</biblScope><biblScope unit="page" from="979">979</biblScope><biblScope unit="page" to="984">984</biblScope></imprint></monogr><idno type="istex">1C47B5A753F389B1E5FBB240B080FCD15C2ED9FF</idno><idno type="DOI">10.1111/j.1540-8159.1984.tb05648.x</idno><idno type="ArticleID">PACE979</idno></biblStruct></sourceDesc></fileDesc><profileDesc><creation><date>1984</date></creation><langUsage><language ident="en">en</language></langUsage><abstract xml:lang="en"><p>The spatial vectorcardiograms (VCG) of 13 patients with WPW syndrome due to single accessory pathways were analyzed and correlated with the excitation analysis obtained on epicardial mapping. The azimuth angle of the initial 10 ms cardiac vector was > + 90° (directed right and anteriorly) in patients with a left ventricular free wall; it ranged between 0° to 90° (left and anteriorly) in those with a left or right paraseptal free wall and was ‐ 30° (left and posteriorly) in one patient with a right ventricular free wall location. The elevation angle of the initial 10 and 20 ms cardiac vector was either zero or positive (inferiorly directed) in those with right and left ventricular free wall pathway. Among six patients with a paraseptal location, the elevation angle was negative (superiorly directed) in four and positive in two. Both the patients with a clockwise inscription of a QRS loop in the horizontal plane (HP) had pathways located to the left ventricle. Among the paraseptal group, at surgery, the accessory pathway could not be excised in two in spite of dissection very close to the IV (interventricular) septum. The elevation angle in both these patients was markedly negative (‐45° and ‐62°) in contrast to the other in whom surgical excision was successful. We thus conclude: 1) an azimuth angle of the initial 10 ms vector of more than 90° indicates early activation in the left ventricular free wall and less than 0° on the right ventricular free wall; 2) presence of a negative elevation angle [superiorly directed) of the initial 10 and 20 ms vector suggests a paraseptal location; 3) a markedly negative elevation angle may further indicate that the accessory connection is located very close to, or in, the IV septum.</p></abstract><textClass xml:lang="en"><keywords scheme="keyword"><list><head>Keywords</head><item><term>spatial vectorcardiography</term></item><item><term>epicardial mapping</term></item><item><term>accessory pathway</term></item></list></keywords></textClass></profileDesc><revisionDesc><change when="1984-11">Published</change></revisionDesc></teiHeader></istex:fulltextTEI><json:item><original>false</original><mimetype>text/plain</mimetype><extension>txt</extension><uri>https://api.istex.fr/document/1C47B5A753F389B1E5FBB240B080FCD15C2ED9FF/fulltext/txt</uri></json:item></fulltext><metadata><istex:metadataXml wicri:clean="Wiley, elements deleted: body"><istex:xmlDeclaration>version="1.0" encoding="UTF-8" standalone="yes"</istex:xmlDeclaration><istex:document><component version="2.0" type="serialArticle" xml:lang="en"><header><publicationMeta level="product"><publisherInfo><publisherName>Blackwell Publishing Ltd</publisherName><publisherLoc>Oxford, UK</publisherLoc></publisherInfo><doi origin="wiley" registered="yes">10.1111/(ISSN)1540-8159</doi><issn type="print">0147-8389</issn><issn type="electronic">1540-8159</issn><idGroup><id type="product" value="PACE"></id><id type="publisherDivision" value="ST"></id></idGroup><titleGroup><title type="main" sort="PACING CLINICAL ELECTROPHYSIOLOGY">Pacing and Clinical Electrophysiology</title></titleGroup></publicationMeta><publicationMeta level="part" position="11006"><doi origin="wiley">10.1111/pace.1984.7.issue-6</doi><numberingGroup><numbering type="journalVolume" number="7">7</numbering><numbering type="journalIssue" number="6">6</numbering></numberingGroup><coverDate startDate="1984-11">November 1984</coverDate></publicationMeta><publicationMeta level="unit" type="article" position="0097900" status="forIssue"><doi origin="wiley">10.1111/j.1540-8159.1984.tb05648.x</doi><idGroup><id type="unit" value="PACE979"></id></idGroup><countGroup><count type="pageTotal" number="6"></count></countGroup><titleGroup><title type="tocHeading1">ORIGINAL ARTICLES</title></titleGroup><eventGroup><event type="firstOnline" date="2006-06-30"></event><event type="publishedOnlineFinalForm" date="2006-06-30"></event><event type="xmlConverted" agent="Converter:BPG_TO_WML3G version:2.3.2 mode:FullText source:HeaderRef result:HeaderRef" date="2010-03-02"></event><event type="xmlConverted" agent="Converter:WILEY_ML3G_TO_WILEY_ML3GV2 version:3.8.8" date="2014-02-06"></event><event type="xmlConverted" agent="Converter:WML3G_To_WML3G version:4.1.7 mode:FullText,remove_FC" date="2014-11-03"></event></eventGroup><numberingGroup><numbering type="pageFirst" number="979">979</numbering><numbering type="pageLast" number="984">984</numbering></numberingGroup><correspondenceTo>Address for reprints: S. Bertil Olsson, M.D., Division of Cardiology, Dept. of Medicine I, Sahlgrenska Hospital, S‐413 45 Göteborg, Sweden</correspondenceTo><linkGroup><link type="toTypesetVersion" href="file:PACE.PACE979.pdf"></link></linkGroup></publicationMeta><contentMeta><unparsedEditorialHistory>Received November 8, 1983; revision received March 1, 1984; accepted March 26, 1984.</unparsedEditorialHistory><countGroup><count type="referenceTotal" number="19"></count><count type="linksCrossRef" number="6"></count></countGroup><titleGroup><title type="main">Spatial Vectorcardiography in the Wolff‐Parkinson‐White Syndrome: Correlation with Epicardial Mapping Findings</title></titleGroup><creators><creator creatorRole="author" xml:id="cr1" affiliationRef="#a1"><personName><givenNames>KEWAL K.</givenNames><familyName>TALWAR</familyName></personName></creator><creator creatorRole="author" xml:id="cr2" affiliationRef="#a1"><personName><givenNames>PER</givenNames><familyName>BLOMSTRÖM</familyName></personName></creator><creator creatorRole="author" xml:id="cr3" affiliationRef="#a1"><personName><givenNames>NILS</givenNames><familyName>EDVARDSSON</familyName></personName></creator><creator creatorRole="author" xml:id="cr4" affiliationRef="#a2"><personName><givenNames>GÖRAN</givenNames><familyName>WILLIAM‐OLSSON</familyName></personName></creator><creator creatorRole="author" xml:id="cr5" affiliationRef="#a1" corresponding="yes"><personName><givenNames>S. BERTIL</givenNames><familyName>OLSSON</familyName></personName></creator></creators><affiliationGroup><affiliation xml:id="a1" countryCode="SE"><unparsedAffiliation>Division of Cardiology, Med. Clin. I, S‐413 45 Göteborg, Sweden</unparsedAffiliation></affiliation><affiliation xml:id="a2" countryCode="SE"><unparsedAffiliation>Division of Thoracic Surgery, Sahlgrenska Hospital, S‐413 45 Göteborg, Sweden</unparsedAffiliation></affiliation></affiliationGroup><keywordGroup xml:lang="en"><keyword xml:id="k1">spatial vectorcardiography</keyword><keyword xml:id="k2">epicardial mapping</keyword><keyword xml:id="k3">accessory pathway</keyword></keywordGroup><abstractGroup><abstract type="main" xml:lang="en"><p>The spatial vectorcardiograms (VCG) of 13 patients with WPW syndrome due to single accessory pathways were analyzed and correlated with the excitation analysis obtained on epicardial mapping. The azimuth angle of the initial 10 ms cardiac vector was > + 90° (directed right and anteriorly) in patients with a left ventricular free wall; it ranged between 0° to 90° (left and anteriorly) in those with a left or right paraseptal free wall and was ‐ 30° (left and posteriorly) in one patient with a right ventricular free wall location. The elevation angle of the initial 10 and 20 ms cardiac vector was either zero or positive (inferiorly directed) in those with right and left ventricular free wall pathway. Among six patients with a paraseptal location, the elevation angle was negative (superiorly directed) in four and positive in two. Both the patients with a clockwise inscription of a QRS loop in the horizontal plane (HP) had pathways located to the left ventricle. Among the paraseptal group, at surgery, the accessory pathway could not be excised in two in spite of dissection very close to the IV (interventricular) septum. The elevation angle in both these patients was markedly negative (‐45° and ‐62°) in contrast to the other in whom surgical excision was successful. We thus conclude: 1) an azimuth angle of the initial 10 ms vector of more than 90° indicates early activation in the left ventricular free wall and less than 0° on the right ventricular free wall; 2) presence of a negative elevation angle [superiorly directed) of the initial 10 and 20 ms vector suggests a paraseptal location; 3) a markedly negative elevation angle may further indicate that the accessory connection is located very close to, or in, the IV septum.</p></abstract></abstractGroup></contentMeta><noteGroup><note xml:id="n-fnt-1" numbered="no"><p>Supported with grants from the Swedish National Society against Heart and Chest Diseases.</p></note></noteGroup></header></component></istex:document></istex:metadataXml><mods version="3.6"><titleInfo lang="en"><title>Spatial Vectorcardiography in the Wolff‐Parkinson‐White Syndrome: Correlation with Epicardial Mapping Findings</title></titleInfo><titleInfo type="alternative" contentType="CDATA" lang="en"><title>Spatial Vectorcardiography in the Wolff‐Parkinson‐White Syndrome: Correlation with Epicardial Mapping Findings</title></titleInfo><name type="personal"><namePart type="given">KEWAL K.</namePart><namePart type="family">TALWAR</namePart><affiliation>Division of Cardiology, Med. Clin. I, S‐413 45 Göteborg, Sweden</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">PER</namePart><namePart type="family">BLOMSTRÖM</namePart><affiliation>Division of Cardiology, Med. Clin. I, S‐413 45 Göteborg, Sweden</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">NILS</namePart><namePart type="family">EDVARDSSON</namePart><affiliation>Division of Cardiology, Med. Clin. I, S‐413 45 Göteborg, Sweden</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">GÖRAN</namePart><namePart type="family">WILLIAM‐OLSSON</namePart><affiliation>Division of Thoracic Surgery, Sahlgrenska Hospital, S‐413 45 Göteborg, Sweden</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">S. BERTIL</namePart><namePart type="family">OLSSON</namePart><affiliation>Division of Cardiology, Med. Clin. I, S‐413 45 Göteborg, Sweden</affiliation><description>Correspondence: Address for reprints: S. Bertil Olsson, M.D., Division of Cardiology, Dept. of Medicine I, Sahlgrenska Hospital, S‐413 45 Göteborg, Sweden</description><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre type="article" displayLabel="article"></genre><originInfo><publisher>Blackwell Publishing Ltd</publisher><place><placeTerm type="text">Oxford, UK</placeTerm></place><dateIssued encoding="w3cdtf">1984-11</dateIssued><edition>Received November 8, 1983; revision received March 1, 1984; accepted March 26, 1984.</edition><copyrightDate encoding="w3cdtf">1984</copyrightDate></originInfo><language><languageTerm type="code" authority="rfc3066">en</languageTerm><languageTerm type="code" authority="iso639-2b">eng</languageTerm></language><physicalDescription><internetMediaType>text/html</internetMediaType><extent unit="references">19</extent></physicalDescription><abstract lang="en">The spatial vectorcardiograms (VCG) of 13 patients with WPW syndrome due to single accessory pathways were analyzed and correlated with the excitation analysis obtained on epicardial mapping. The azimuth angle of the initial 10 ms cardiac vector was > + 90° (directed right and anteriorly) in patients with a left ventricular free wall; it ranged between 0° to 90° (left and anteriorly) in those with a left or right paraseptal free wall and was ‐ 30° (left and posteriorly) in one patient with a right ventricular free wall location. The elevation angle of the initial 10 and 20 ms cardiac vector was either zero or positive (inferiorly directed) in those with right and left ventricular free wall pathway. Among six patients with a paraseptal location, the elevation angle was negative (superiorly directed) in four and positive in two. Both the patients with a clockwise inscription of a QRS loop in the horizontal plane (HP) had pathways located to the left ventricle. Among the paraseptal group, at surgery, the accessory pathway could not be excised in two in spite of dissection very close to the IV (interventricular) septum. The elevation angle in both these patients was markedly negative (‐45° and ‐62°) in contrast to the other in whom surgical excision was successful. We thus conclude: 1) an azimuth angle of the initial 10 ms vector of more than 90° indicates early activation in the left ventricular free wall and less than 0° on the right ventricular free wall; 2) presence of a negative elevation angle [superiorly directed) of the initial 10 and 20 ms vector suggests a paraseptal location; 3) a markedly negative elevation angle may further indicate that the accessory connection is located very close to, or in, the IV septum.</abstract><subject lang="en"><genre>Keywords</genre><topic>spatial vectorcardiography</topic><topic>epicardial mapping</topic><topic>accessory pathway</topic></subject><relatedItem type="host"><titleInfo><title>Pacing and Clinical Electrophysiology</title></titleInfo><genre type="Journal">journal</genre><identifier type="ISSN">0147-8389</identifier><identifier type="eISSN">1540-8159</identifier><identifier type="DOI">10.1111/(ISSN)1540-8159</identifier><identifier type="PublisherID">PACE</identifier><part><date>1984</date><detail type="volume"><caption>vol.</caption><number>7</number></detail><detail type="issue"><caption>no.</caption><number>6</number></detail><extent unit="pages"><start>979</start><end>984</end><total>6</total></extent></part></relatedItem><identifier type="istex">1C47B5A753F389B1E5FBB240B080FCD15C2ED9FF</identifier><identifier type="DOI">10.1111/j.1540-8159.1984.tb05648.x</identifier><identifier type="ArticleID">PACE979</identifier><recordInfo><recordContentSource>WILEY</recordContentSource><recordOrigin>Blackwell Publishing Ltd</recordOrigin></recordInfo></mods></metadata><serie></serie></istex></record>Pour manipuler ce document sous Unix (Dilib)
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